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One proposal is that hypofunction of NMDA receptors (NMDAR) located on inhibitory GABA inter-neurons leads to reduced activity of these inter-neurons, resulting in increased pyramidal glutamatergic neurotransmission and the diverse symptoms occurring in acute psychosis [3]

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Repeated-dose oral N-acetylcysteine in Parkinsons disease: pharmacokinetics and effect on brain glutathione and oxidative stress

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