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The data was processed with RTCA Pro software of RTCA eSight Software v1.3 (Agilent)

Notably, as suggested in an animal model of neuroinflammation induced by ischemic stroke damage known to increase the prevalence of M1 microglia in the brain, pre-treatment with melatonin upregulated the expression of TREM2 while downregulating that of proinflammatory genes in microglia (e.g., inducible nitric oxide synthase) [101], suggesting that melatonin may polarize microglia towards its M2 phenotype

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On the other hand, GHK-Cu promotes angiogenesis, the formation of new blood vessels, which is a process that tumors exploit to fuel their growth