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Aos, upon binding to cell-surface receptors on neurons, disrupt signal transduction, leading to a sustained interruption of intracellular Ca 2+ signaling through increased N-methyl-D-aspartate (NMDA) receptor-dependent Ca 2+ influx [77]
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Available at: (Accessed on 16th December 2025)
The plasma and erythrocyte ChE activities of the monkeys decreased to approximately 50% of pre-exposure levels, but rapid recovery took place after cessation of exposure
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