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A growing body of evidence emphasizes the pivotal role of inflammation in the progression of HACE (7, 28)

Genetic profiling can help in doing targeted interventions by identifying risk-related genes like the APOE 4 variant

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Older neutrophils are at a higher risk of apoptosis due to a lower responsiveness to factors such as G-CSF, IL-2, or tumor necrosis factor (TNF)- that would normally prevent them from undergoing programmed cell death [130, 131]

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