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Because we proposed that ASM would evaluate performance at an individual clinician level (TIN/NPI), TIN alone would not be useful for ASM

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In endothelial cell models, BPC-157 significantly enhanced ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formation mediated through downstream activation of transcription factors c-Fos, c-Jun, and EGR-1

Related work showed context-appropriate angiogenesis during muscle/tendon healing, with VEGF-linked markers in vivo (Brcic 2009)