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Notably, researchers observed that the rise in p21 levels, a marker of senescence, was independent of p53, indicating that, unlike many previous drugs, AKI603-induced senescence may operate independently of p53 activity [79]

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Furthermore, by employing the MPTP mouse model to simulate mitochondrial damage and GSH deficiency in PD, we demonstrate that pyridoxine-induced GSH enhancement helpes to reduce the severity of dopaminergic neuron loss and improve some types of mouse locomotion after MPTP administration