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236 , R109R143 (2018)

These techniques satisfy the necessity of the identification of optimal targets for the so called redox gene therapy that has been already demonstrated to overcome problems associated to oxidative damage during I/R after liver transplantation.48 Therefore, rather than delivering through transgene vectors, classical oxidant scavengers, such as SOD or CAT, the overexpression of early genes responsive to oxidative stress could be most efficient in protecting cells from acute doses of ROS and would provide an optimal tool to face the majority of liver pathologies

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These guideline changes can influence practice patterns without requiring system-level modifications (Practice Committee of the American Society for Reproductive Medicine, 2014)