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While some degree of lean mass decline is physiologically expected during negative energy balance, excessive or disproportionate reductions in skeletal muscle may have important clinical implications in individuals with limited anabolic reserve, defined as a reduced capacity to maintain or restore skeletal muscle mass due to diminished anabolic hormone availability and/or impaired responsiveness of muscle tissue to anabolic stimuli such as testosterone [6,7]

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The studies outlined here identify a specific pro-oxidant pathway that dramatically impacts both intra- and extracellular redox states, enhances T2 inflammation and cell death, and is associated with worsening asthma control