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Patients with type 2 diabetes can be broadly partitioned into those with predominantly inflammatory-driven insulin resistance characterized by elevated CRP, IL-6, and TNF-, higher visceral adiposity, and greater macrovascular risk and those whose disease is driven principally by beta-cell insufficiency with a less prominent inflammatory signature

Higher doses were more impactful in decreasing the oxidized to reduced glutathione ratio, decreasing biomarkers of oxidative stress, and improving immune function, lymphocyte proliferation, and natural killer cell activity

Increasing too quickly can generate more side effects