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Endoplasmic reticulum calcium release through ITPR2 channels leads to mitochondrial calcium accumulation and senescence

Poster Session II - NOVEL TARGET/ EXPERIMENTAL FIDINGS TO CLINICAL TRANSLATION 06.00 - NOVEL TARGET/ EXPERIMENTAL FIDINGS TO CLINICAL TRANSLATION - 06.01 - PATHOPHYSIOLOGY OF STROKE / TRANSLATIONAL MEDICINE P1080 - ESOC25-706 INFLAMMATORY BLOOD CELL COUNTS PREDICT HEMORRHAGIC TRANSFORMATION AND 3-MONTH DISABILITY IN ISCHEMIC STROKE PATIENTS, WITH AND WITHOUT ACUTE REVASCULARIZATION TREATMENTS Julien Mouyal 1 , Patrik Michel 1 , Davide Strambo 1 1 Lausanne University Hospital, Lausanne, Switzerland Background and Aims: The inflammatory response and immune regulation are critical factors for ischemic brain injury

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The research suggesting that lower GH correlates with longer lifespan in animal models applies to sustained, non-physiological GH elevation, not to short-burst pulsatile release that restores a more youthful GH pattern