arsenic glutathione

arsenic glutathione Nrf2 activation ameliorates cytotoxic effects of trioxide in acute promyelocytic leukemia cells through increased levels and arsenic efflux from cells Frontiers | Exogenous Glutathione Increases

Name: arsenic glutathione Nrf2 activation ameliorates cytotoxic effects of trioxide in acute promyelocytic leukemia cells through increased levels and arsenic efflux from cells Frontiers | Exogenous Glutathione Increases
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Description

Hanania E, Fu S, Roninson I, Mechetner E, Hegewisch-Becker S, Andreeff M , Berenson R, Heimfeld S, Gottesman M, Deisseroth A

Alterations in both pathways enhance oxidative stress and increase APAP toxicity

Mechanisms of copper-induced toxicity include induction of apoptosis (181-183), caspase-independent cell death pathways (184-186), induction of ROS or inhibition of the ubiquitin-proteasome system (129,187-189)