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however, the aging brain exhibits heightened basal levels of proinflammatory cytokines such as interleukin-6 (IL-6), as well as a disequilibrium of anti-inflammatory signals, resulting in activation of CNS-resident microglia (Pachter et al., 2003

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An increasing body of evidence suggests that the KD, owing to its metabolic and biochemical properties, may have therapeutic potential in a range of neurological disorders beyond its established applications [8]

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Why injection site doesn't matter for TB-500 (unlike BPC-157): TB-500 circulates through the bloodstream and migrates to areas of tissue damage - it seeks out injured tissue via actin upregulation signals