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In endothelial cells (ECs), copper overload disrupts mitochondrial dynamics, increases fission and ROS production, and ultimately leads to cell death [34] In cardiomyocytes, copper promotes hypertrophy and inflammation [35], disrupts fatty acid and lipid metabolism, upregulates autophagy-associated proteins and genes, and impedes calcium ion uptake by the sarcoplasmic reticulum, thereby inhibiting myocardial contractility [36]

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Iron Balance and Cardiovascular Health: The Double-Edged Role of Deficiency and Overload

Bioavailability refers to how well your body can actually absorb and use a substance after you take it

pylori GuaB and compared the activity of a previously tested benzimidazole-based inhibitor with newly synthetized indole-based small molecules