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Decreased mitochondrial fatty acid oxidation of SLC25A45 KO mice is not a consequence of general mitochondrial dysfunction per se for the following reasons: First, SLC25A45 KO mitochondria exhibited equivalent oxygen consumption rate (OCR) to control mitochondria when pyruvate, glutamine, and succinate were provided ( Fig

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These concerns may be magnified by the fact that a very young childs liver may still be developing and thus a childs ability to metabolize the drug may be limited, the letter stated