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In cellular (HT-22 cell) experiments, PCHP attenuated reactive oxygen species (ROS) generation induced by lipopolysaccharide (LPS) ( CHP reversed these changes, and decreased proinflammatory cytokine secretion ( In addition, PCHP treatment counteracted the decreased levels of GluA1, HO-1, activity-regulated cytoskeleton-associated protein, phospho-tyrosine kinase receptor B, Nrf2, postsynaptic density protein 95, and brain-derived neurotrophic factor ( 5.2 Mechanisms underlying the biological activities 5.2.1 Antioxidant mechanism PCHP exhibit antioxidant activity primarily by modulating the Keap1-Nrf2-ARE signaling pathway

Bottom-up analysis of emergent properties of N-acetylcysteine as an adjuvant therapy for COVID-19

Shu, L., Murphy, H

Indian J Ophthalmol 59(Suppl1):S97S101 Lei H-T et al (2023) LncRNA-mediated cell autophagy: an emerging field in bone destruction in rheumatoid arthritis