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The peptide maintained protective effects throughout natural lifespan without evidence of tolerance or diminishing efficacy

Six models were fit with BrAC, perceived drunkenness, alcohol craving, appetite, blood glucose, and nausea scores as dependent variables, respectively (Supplemental Table 2)

We used a lag of 6 months for all exposures to minimise protopathic bias and allow for a minimum and sufficient latency period after cohort entry [17, 18]