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This cellular mechanism provides a biological basis for how PQQ may help enhance mitochondrial density and function, directly correlating with efforts to support cellular energy and reduce fatigue

Qiu et al., 2016), radioactive skin damage (Kong et al., 2016

In the streptozotocin (STZ)-induced diabetic rat model, intravitreal injection of the ferroptosis inhibitor Fer-1 significantly reduced retinal inflammation, maintained the integrity of BRB, and prevented RGCs loss, which was confirmed 48 weeks after diabetes induction (232, 233)

Its core functions are mainly reflected in three aspects: Energy Metabolism: NAD+ participates in the process by which mitochondria convert food into usable cellular energy (ATP), which is fundamental to maintaining life activities

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