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The STEP-HFpEF trial further supports semaglutide potential to prevent HFpEF progression by reducing pathological ventricular remodeling through significant weight reduction [9,13]

Off-label use in younger patients occurs in clinical practice, but such decisions require careful provider assessment of risk-benefit, family history, and metabolic status

C32 and C88 in PfGrx) could not only form an intramolecular disulfide during catalysis or affect the reactivity of the active-site thiolate but also function as a resolving cysteine for trapped Grx(SSR) species (Fig

The study population may have differed in meaningful ways

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