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Phenylbutazone, Enoxacin, and Doripenem hydrate were dosed at 50, 0.3, and 26 mg/kg, with N = 46 per group, which is sufficiently powered based on prior studies 50,51,52
Meta-analysis was conducted using a random effects model where heterogeneity was low to moderate (I 2 = 0-60%)
Fatty acid oxidation favored the polarization of macrophages towards a resolution/repair profile in response to efferocytosis of ACs or IL-4 stimuli, with lipin-1 activity being critical for establishing a reparative and pro-resolution phenotype in macrophages in the atherosclerotic plaques [76]
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