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659 The level of GPX4 in the brain gradually decreases after TBI, and overexpression of GPX4 can partially reverse TBI-induced hippocampal ferroptosis and synaptic damage, effectively alleviating neuronal dysfunction after cerebral hemorrhage, and improving TBI-induced cognitive deficits
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On this background, a synthetic replica of exendin-4, exenatide, was developed for clinical T2DM therapy with two daily injections (110)
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