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These findings highlight caudatins potential to target the host-virus interface at the transcriptional level, offering a novel strategy to circumvent nucleoside resistance in HBV therapy (Figure 4B)

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A recent study reported that the novel mitophagy inducer miriplatin-loaded liposomes (LMPt) enhance LONP1 binding to polymerase gamma (POLG) and TFAM, thereby resulting in POLG and TFAM degradation, which inhibits mtDNA replication and suppresses pancreatic cancer cell proliferation [160]