Despite sharing the five HF-TAFs, TFIID and SAGA architectural scaffold form distinct structures mostly due to the incorporation of alternative interaction partners (6, 7) Intriguingly, many TAFs exist as paralogs or are expressed in multiple isoforms, which differ in sequence, structure, and tissue distribution leading to formation of alternate TFIID-like complexes and modulating transcription in a cell-type-specific, developmental stage-specific, or signal-dependent manner (11-13)
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Angiotensinogen converts angiotensin I to angiotensin II, which exerts its effects via angiotensin II type 1 receptor (AT 1 R) [149]
