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FOXO4-p53 Disruption Moderate Research Detailed mechanistic studies confirm that FOXO4-DRI achieves its senolytic effects through precise disruption of the protective FOXO4-p53 protein interaction that allows senescent cells to evade apoptosis and persist in tissues

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Interestingly, CagriBB exhibited a reduced potency relative to Cagri in cells expressing CTR alone (Supplementary Fig