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in colorectal cancer, tumor-derived lactate promotes macrophage H3K18la to repress RAR transcription and elevate IL-6, which activates STAT3/c-Myc signaling in cancer cells to reinforce M2-like TAM polarization ( Studies have shown that Kla modification sites exist in TAMs, and Kla levels correlate with macrophage transition to the M2 phenotype

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