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MTZ attenuates Cd-induced neurotoxicity by upregulating the transcription factor Nrf2, suppressing NF-B/TLR4 signalling, and effectively decreasing TNF-, IL-1, and IL-6

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Given that -MSH [or its larger parent peptide adrenocorticotropic hormone (ACTH)] represents the only known melanocortin ligand, whereas -endorphin is one of multiple endogenous ligands capable of activating -opioid receptor signaling, a decrease in both -MSH and -endorphin might result in a net increase in opioid signaling sustained by nonPOMC-derived opioid ligands