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Research shows that in senescent cells, p53 levels are maintained low through ubiquitination while its pro-apoptotic activity is repressed through nuclear segregation by FOXO4. The peptide's competitive inhibition restores p53 activity by promoting nuclear exclusion, allowing p53 to execute its pro-apoptotic program and eliminate senescent cells through targeted pathway activation

BMJ , 326(7399), 1124

Influence of dietary habits on oxidative stress markers in Hashimotos thyroiditis

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Most of our understanding of this multistep pathway stems from studies in yeast and bacteria, where the sequential actions of these enzymes have been genetically and biochemically dissected 28,33