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The DksA-dependent regulatory profile that is independent of (p)ppGpp and connected to ROS/RNS stress down-regulates both the ribosome biogenesis machinery and amino acid biosynthesis, while also promoting redox buffering and repair of biomolecules, thus resulting in broad cellular resistance to these conditions

Targeted therapy resistance, a major clinical challenge, is amenable to intervention via the autophagy-ferroptosis axis, a key determinant of cell fate under stress, through two primary strategies

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Brown E, et al

This inconsistency can affect both efficacy and side effects, as the body repeatedly adjusts to changing medication levels