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These recent experimental findings are biologically plausible, particularly as both oxidative stress and neuroinflammation are increasingly recognized as important contributors to the pathophysiology of ASD (151, 156, 160)

Poses that significantly deviated from the binding pose of L -tyrosine were flagged asunreasonable poses

Wood SJ,Ycel M,Pantelis C,Berk M, Neurobiology of schizophrenia spectrum disorders: the role of oxidative stress

The role of cellular senescence in neurodegenerative diseases