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These protective responses have been associated with coordinated biological actions involving vascular support, regenerative signaling, and regulation of inflammatory activity, collectively contributing to enhanced tissue resilience and recovery in injury models

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This process promotes ubiquitination and degradation of Toll-like receptor 4 (TLR4), thereby inhibiting the NF-B signaling pathway and reducing IL-1 expression in M1 macrophages, ultimately alleviating lung inflammatory responses (90)