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To further develop our understanding of the impact of plasma treatment on GSH and GSSG as the central cellular redox system, we utilized the iron(II) complex ferrocenecarboxylic acid (complex 1 ) and the iron(III) complexes iron(III) acetylacetonate (Fe(acac) 3 ) (complex 2 ) and chloro(protoporphyrinato)iron(III) (hemin) (complex 3 Fig

Isotretinoin metabolites, particularly 4-oxo-isotretinoin, also raise hepatic ROS and deplete glutathione stores [8]

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