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Abstract Hypobaric hypoxia is widely recognized as a prominent risk factor for high-altitude cerebral edema (HACE), which contributes to the exacerbation of multiple pathological mechanisms, including oxidative stress, mitochondrial dysfunction, disruption of blood;brain barrier integrity, neuroinflammation, and neuronal apoptosis

Huntingtons disease: a clinical review

Conversely, treatment with free DOX resulted in significant cell death in normal organs, particularly the heart, while the toxicity of DOX/LAAM GSH-CQDs was minimal (Supplementary Fig

Treatment of AS mice with P-Mn/TI@mHSA The prophylactic and therapeutic efficacy of P-Mn/TI@mHSA against atherosclerotic plaques was evaluated

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