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Notably, miR-155 and miR-132 are among the dysregulated miRNAs implicated in key pathogenic pathways, including the NF-B signaling pathway, oxidative stress responses, and mitochondrial-mediated apoptosis, all of which contribute to testicular dysfunction and impaired spermatogenesis [14, 15]

Finally, BMIS individuals harboring CAG01909 had significantly higher circulating TMAO than those that lacked it (Supplementary Fig

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