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(133) indicated that dietary interventions rich in dietary fiber and fermented foods have the potential to increase gut microbiota diversity and reduce the levels of inflammatory markers (133)

Here, we summarize the promising therapeutic targets for SLE therapy, which align with recent developments in understanding the diseases pathogenesis and the diversification of the SLE drug pipeline

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These alterations coincide with higher levels of Beclin-1, LC3B-II, and SQSTM1 and contribute to neuroinflammation and neurodegeneration [88, 89]

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