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Since IL-17 receptor A (IL-17RA) has been shown to mediate communication from the meninges to the cortex 47 , we next examined whether the beneficial effect of peripherally targeting CD38 could be mediated in part through modulation of cortical IL-17RA and IL-1 signaling, which we observed to drive T H 17 pathogenicity

10.1021/ol0501626

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Moreover, investigations within a porcine myocardial infarction model have shown that MCC950 notably diminishes infarct size and alleviates cardiomyocyte pyroptosis through the inhibition of NLRP3 inflammasome activation, consequently reducing the release of inflammatory factors (IL1, IL18)