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Moreover, activation of endothelial nitric oxide synthase (NOS) in HUVECs by GIP at 1 nM decreased inflammatory inducible NOS expression levels

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While the specific roles and interactions of direct viral mechanisms, indirect effects mediated by the host immune response, and these other contributing factors in NeuroHIV pathogenesis remain complex and not fully elucidated, growing evidence increasingly highlights the central role of microglial activation and subsequent neuroinflammation

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