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An emerging area of interest lies in the role of cholesterol metabolism in the pathogenesis of NAFLD

This mechanism effectively suppresses the assembly of the inflammasome and the subsequent secretion of IL1 and IL18

HPV oncoproteins disrupt mitochondrial function, leading to OS and DNA damage, promoting carcinogenesis

In fact, studies have also shown that increased inflammation and oxidative stress may result in higher concentrations of HPV in the cervical and vaginal secretions which then make the cervical cells more vulnerable to cellular damage

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