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Xu, L., et al

The novel DA-CH3 dual incretin restores endoplasmic reticulum stress and autophagy impairments to attenuate Alzheimer-like pathology and cognitive decrements in the APPSWE/PS1DeltaE9 mouse model

Its groundbreaking mechanism that stimulates both GIP and GLP-1 receptors represents an innovative method of treatment, offering a dual-approach method that distinctively supports blood sugar regulation

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This is consistent with previous reports demonstrating that erastin promotes ferroptosis in ihESCs and attenuates EMs progression [29, 30]