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Several experimental agents and strategies are now being developed for thromboinflammatory, autoimmune, and metabolic diseasesand are conceptually applicable to PCOS
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Bioinformatics analysis indicated that the riboflavin deficiency caused an up-regulation of Parkinsons disease pathway, steroid catabolism, endoplasmic reticulum stress and apoptotic process, while the fatty acid metabolism, tricarboxylic citrate cycle, oxidative phosphorylation and iron metabolism were down-regulated