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In fact, many studies have reported the eryptotic effect of bile acids and bilirubin on RBCs, whereas albumin reduces the toxic effect of bilirubin on erythrocytes [210, 283]

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While crosstalk between ferroptosis and cuproptosis has been suggested, direct evidence identifying the key molecular integrators (e.g., how GSH depletion precisely tilts the balance between the two) and the ultimate cell fate decisions within a relevant TME is scarce

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The clinical spectrum of chronic metabolic acidosis: homeostatic mechanisms produce significant morbidity