Honestly, the best approach for a research model likely incorporates elements of both
In individuals with ASD, reduced GSH levels and disrupted redox balance have been associated with chronic neuroinflammation within the cerebellum and temporal lobe in postmortem brain tissue (Al-Gadani et al., 2009
Temporal profile of matrix metalloproteinases and their inhibitors after spontaneous intracerebral hemorrhage: relationship to clinical and radiological outcome
Bruton's tyrosine kinase is essential for NLRP3 inflammasome activation and contributes to ischaemic brain injury
In fact, by continuously depriving the liver of esterified fatty acids and inhibiting the hepatic glycolytic pathway, glucagon promotes the production of ketone bodies [41,42,43] Hepatic ketogenesis is boosted as a result, and mitochondrial fatty acid oxidation is improved [14, 43]