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Neuroprotection through TNFR1 appears to involve both the induction of survival-related genes and caspase-dependent apoptosis when survival cues are insufficient [46]

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144 In TNBC tissue, WTAP, NUPR1, and LCN2 are significantly overexpressed, and WTAP upregulates LCN2 expression by regulating NUPR1 m6A modification