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SNIP1 mutations in the FHA domain affect cell viability and pre-mRNA splicing To determine whether mutations in the SNIP1 FHA domain affect incorporation of SNIP1 into the activated spliceosome, we expressed flag-tagged SNIP1 (F-SNIP1) WT, single mutants K265G, K301G, R365G, E366G, and the double mutant K265G/K301G in cells and compared their association with components of the activated spliceosome (Fig

(10) Studies have documented that IGF-1 signaling promotes fibroblast proliferation, collagen synthesis, and angiogenesisall critical components of wound healing

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