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Impaired magnesium ion binding and carbohydrate kinase activities corresponded with dysfunctional RBKS and PRTFDC1, indicating TCA cycle disruption and mitochondrial oxidative phosphorylation inefficiency [63, 64]

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Treatment with 5-Amino-1MQ combined with diet transition produced dramatic whole-body adiposity and weight loss, rapidly normalising these measures to levels comparable to age-matched lean control animals an outcome that diet switch alone was unable to achieve in the same time frame

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