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Furthermore, cells in several types of cancers are characterized by significant enhancement of glutathione biosynthesis, and this metabolic vulnerability has been targeted to sensitize these cancer cells to ROS-induced drugs 63

Transcriptional activation of Chac1 and other Atf4-target genes induced by extracellular l-serine depletion is negated with glycine consumption in Hepa1-6 hepatocarcinoma cells

10.1016/j.clinph.2014.05.021 Clin

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