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Therefore, yolk fatty acid composition closely mirrors the lipid profile of hepatic VLDLy and VTG, providing a mechanistic explanation for the rapid and dose-responsive modulation of yolk -3 PUFAs levels following dietary intervention
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When glutathione is depleted, the mitochondria sustain physical damage from their own energy production processes, leading to a catastrophic drop in ATP output
Moreover, ROS can impair the epithelial barrier by disrupting the mucus layer, a protective barrier on the intestinal epithelial surface composed of mucins such as MUC2 secreted by IECs
Thus, it is tempting to attribute an increase in lactate to an imbalance between glycolysis and mitochondrial oxidation, similar to, but less severe than hypoxia
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