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2.1.2 Vesicular monoamine transporter 2 inhibition METH inhibits vesicular monoamine transporter 2 and disrupts monoamine storage in synaptic vesicles (Lizarraga et al., 2015)
Given their pivotal roles, targeting mitochondrial metabolism and copper homeostasis presents a promising strategy for novel therapeutic interventions in several cardiovascular diseases
Expression and activation of matrix metalloproteinases in wounds: modulation by the tripeptide-copper complex glycyl-l-histidyl-l-lysine-Cu2+
Drugdrug and drugdisease interactions OTC sympathomimetics may potentiate an acute hypertensive effect
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