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Extensive research confirms that it effectively alleviates hepatic steatosis, a process thought to be mediated by the activation of the hepatic AMPK signaling pathway

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In contrast, metabolites such as phosphatidic acid, phosphatidylglycerol (16:0/0:0), PC (16:0/3:1(2E)), and phosphatidylinositol (16:0/16:0) are significantly upregulated, suggesting a high saturation of glycerophospholipids