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As a consequence of AKI, a significant number of oxygen free radicals are produced, endogenous antioxidants are continually depleted, and high levels of inflammatory substances are secreted, all of which contribute to kidney damage progression (Han and Lee, 2019)

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Larger studies incorporating standardized dosing regimens are needed to more clearly establish whether NAC is useful as a preventive or treatment agent for these disorders

These results suggest that anti-MSTN therapy may be a potential target for delaying obesity and hyperlipemia

Third, it can trigger inflammation, which can also contribute to hepatocyte death [42]