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Additional conditions under the umbrella of congenital IGF-1 deficiencies include a documented case of a 15-year-old boy suffering from severe prenatal and postnatal growth failure, sensorineural deafness and mental retardation due to a homozygous partial deletion of the IGF-I gene [41] as well as IGF1R genes defects [42]

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NAD + as a therapeutic target in fatty liver disease A synthesis of extant research has demonstrated that NAD + is a therapeutic target that merits further exploration

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