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Particular emphasis is placed on the molecular recognition mechanisms between DAMPs and PRRs (TLRs, NLRs, CLRs and RAGE), and the disease regulatory networks formed by activated key signaling pathways (NF-B, MAPK, inflammasomes and cGAS-STING)

[] These analogues, as are displayed in Figure 5, have been shown to possess better excretory efficacy for reducing arsenic and lead intoxications, are capable of crossing biomembranes, have enhanced tissue metal uptake and show lower toxicity (Flora et al ., 2008

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Some Phase I reactions produce electrophiles or free radicals that must be rapidly neutralized through glutathione and other Phase II pathways